THE BASIC PRINCIPLES OF WERNER SYNDROME RECQ HELICASE-IN-1

It's appealing to notice that once the DCM-resulting in mutation K36Q in cTnI was introduced inside the presence of Ca2+, the simulation showed that Ser 22 and 23 no longer interacted closely with cTnC, in accord with our hypothesis that the Ca2+-cTnC-cTnI N terminus conversation is exclusive and is particularly destabilized straight by phosphoryla